If acetylcholinesterase were inhibited at the neuromuscul...

If acetylcholinesterase were inhibited at the neuromuscular junction, the most likely immediate effect would be:

hard

ALess stimulation of the muscle fiber and weaker contraction

Prolonged stimulation of ACh receptors, causing sustained depolarization and muscle over-activation

CNo effect because ACh breakdown is not required for signaling

DInstant destruction of troponin

EOnly the tendon would contract, not the muscle

Explanation

Blocking AChE lets ACh accumulate, keeping receptors activated longer. This prolongs depolarization and can lead to spasms and eventually depolarization block/fatigue.

Previous Question Next Question

Report

Back to Deck — Muscle Contraction, Muscle Tissue & Tendons

Source: entermedschool.org

Related Questions

Related Questions